全部文献期刊学位论文会议报纸专利标准年鉴图书|学者科研项目
中外文文献  中文文献  外文文献
作者:Anna Torres , Joanna Kozak , Agnieszka Korolczuk ...
来源:[J].BMC Cancer(IF 3.333), 2016, Vol.16 (1)Springer
摘要:...(#br)Based on our previous studies we speculated that miR-92a exhibited pro-oncogenic properties in endometrial cancer, and therefore its inhibition could be used as a therapeutic measure in this disease. Therefore in the present study we aimed to investigate both in vitro and...
作者:Liang Wang , Jinhong Wu , Changao Xie
来源:[J].Iranian Journal of Basic Medical Sciences(IF 0.243), 2017, Vol.20 (7), pp.783-790DOAJ
摘要:... However, the function of miR-92a is still unclear in HCC. Materials and Methods: Expression of miR-92a in human HCC cell lines was evaluated using qRT-PCR. MTT assay and transwell assay were used to examine the function of miR-92a in HepG2 and Huh7 cells. Bioinformatic analys...
作者:Shien Hu , Lan Liu , Eugene B. Chang ...
来源:[J].Molecular Cancer(IF 5.134), 2015, Vol.14 (1)Springer
摘要:...(#br) Methods(#br)miR-92a levels were measured in archived human colon cancer and adjacent normal colon specimens by microarray and quantitative RT-PCR (qPCR). The effects of butyrate and other histone deacetylase inhibitors (suberoylanilide hydroxamic acid (SAHA) and valpro...
作者:Mohammadreza Sharifi , Rasoul Salehi , Yousof Gheisari ...
来源:[J].Turkish Journal of Hematology(IF 0.494), 2013, Vol.30 (2), pp.157-162DOAJ
摘要:... In acute myeloid leukemia up-regulation of miR-92a has been reported in humans in vitro studies. In this study it is mainly aimed to assess the effect of inhibition of miR-92a in viability of acute promyelocytic leukemia (APL). METHODS: We performed inhibition of miR-92a in a...
作者:Lan Zhang , Mi Zhou , Yingjie Wang ...
来源:[J].Apoptosis(IF 3.949), 2014, Vol.19 (6), pp.975-983Springer
摘要:... Oxidative stress in diseased vessels promotes VSMC apoptosis in part by activating the c-Jun N-terminal kinase (JNK) pathway, which has been identified as a molecular target of miR-92a in macrophages. Here, we examined the expression and biological activity of miR-92a in V...
作者:Jun Guo , Ning Wen , Sefei Yang ...
来源:[J].Biomedicine & Pharmacotherapy(IF 2.068), 2018, Vol.104, pp.77-86Elsevier
摘要:... However, little is known about the potential role of microRNA-92a (miR-92a) in OSCC. Thus, the aim of this study was to investigate the effects of miR-92a expression on OSCC cell growth, apoptosis and tumorigenesis. Real-time quantitative polymerase chain reaction was used ...
作者:Xuesheng Jiang , Xiongfeng Li , Fengfeng Wu ...
来源:[J].Gene(IF 2.196), 2017Elsevier
摘要:... Among numerous cancer-related miRNAs, the expression level of miR-92a and its potential role in OS has not been investigated. Here, We showed that overexpression of miR-92a was identified in OS specimens and cells compared to normal bone tissues. The high level of miR-92a was...
作者:Miao Lv , Huahui Chen , Yina Shao ...
来源:[J].Fish and Shellfish Immunology(IF 2.964), 2017, Vol.69, pp.211-217Elsevier
摘要:Abstract(#br)miR-92a, a well-documented oncogene, was previously found to be differentially expressed in diseased sea cucumber Apostichopus japonicus by high-throughput sequencing. In this study, we identified Aj14-3-3ζ as a novel target of miR-92a in this species and investiga...
作者:Kai Ma , Xixi Li , Hongxia Hu ...
来源:[J].Comparative Biochemistry and Physiology, Part B(IF 2.069), 2017, Vol.203, pp.20-24Elsevier
摘要:... MiR-92a was found to be overexpressed in a deltamethrin-resistant (DR) strain. The association of miR-92a with pyrethroid-resistance was investigated by quantitative reverse transcription PCR (qRT-PCR). Expression levels of miR-92a were 2.72-fold higher in the DR strain ...
作者:Baozhen Sun , Jingzhe Zhang , Meihan Liu ...
来源:[J].Biomedicine & Pharmacotherapy(IF 2.068), 2019, Vol.114Elsevier
摘要:... Expression of miR-92a in cell was altered by transfection with miR-92a-mimic (miR-92a-M) or miR-92a-inhibitor (miR-92a-I). Cell viability, proliferation, apoptosis, migration and invasion were detected by Cell Counting Kit-8, BrdU assay, flow cytometry, and transwell assay...

我们正在为您处理中,这可能需要一些时间,请稍等。

资源合作:cnki.scholar@cnki.net, +86-10-82896619   意见反馈:scholar@cnki.net

×